Defending season champion Steve Torrence took the Top Fuel qualifying lead in the season-opening Lucas Oil NHRA Winternationals at Auto Club Raceway
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Defending season champion Steve Torrence took the Top Fuel qualifying lead in the season-opening Lucas Oil NHRA Winternationals at Auto Club Raceway

The Haas Formula One team has changed its livery from a metallic gray to a more dynamic black-and-gold for the upcoming season


Stress doesn’t come the same way twice: It’s that fluttering anxiety in your stomach before you enter a dinner party you really didn’t want to go to. It’s that heaviness in your chest as you await the results of a medical test. It’s that bubbling up in your throat as your to-do list gets longer and longer. And it’s even the frazzled, shortness of breath just before a total anxiety meltdown.
The point is, you know each of these situations better than you’d like to. But no matter which kind you’re most acquainted with, one thing rings true: It’s not good for your body, your soul, or your brain. Recent research in British Medical Journal Open finds experiencing extreme stress (such as a divorce, illness, or job loss) increases your risk of Alzheimer’s by 21 percent and any other dementia by 15 percent, bringing truth to the old adage: It’s all in your head.
Although the typical stressors for human beings have long evolved from bears and wolves to finances and relationships, the human body has yet to catch up. When you see your bank account near the red zone, for example, you activate your body’s sympathetic nervous system, or fight-or-flight response. Your heart starts beating faster, cortisol rushes through your body, and oxygen gets pumped to your brain to optimize you for the fight ahead. Except there is no fight—only a frantic run to the ATM. And this happens every time it’s activated. And as research shows, using up all those resources in chronic stress can be taxing on the entire body. Take a closer look at what your body on stress looks like:
You don’t need to experience “extreme” stress in order to see a change in your memory. New research in the Journal of Neuroscience discovered a link between high levels of the stress hormone cortisol and short-term memory loss in aging adults.
Sudden stress might cause you to become temporarily pale after redirecting blood from your face to muscles that might need it. But constant low-grade stress over time can cause your skin to age faster; stir up rashes, rosacea, and psoriasis thanks to a histamine release; and increase oil production (read: acne).
Both chronic stress and repeated traumatic incidents (called acute stress by doctors) lead to inflammation in the coronary arties, which are responsible for heart attacks. The constant uptick of the heartbeat in those with chronic stress also takes a toll on the heart’s ability to pump blood, upping the risk for hypertension and stroke.
Quickened breath is a classic symptom of stress. But if you have asthma, studies show that stress can actually trigger an attack, while those with emphysema could have trouble getting enough oxygen in a stressful situation. Another dig: Stress predisposes you to inflammation, making asthma attacks more common.
Science confirms what women (and men!) everywhere know to be true: Stress fat is real. A study in Biological Psychiatry found women who experienced one or more stressful events the day before eating an indulgent meal burned fewer calories from the meal than those who weren’t stressed. The difference can amount to an extra 11 pounds per year.
Being in constant survival mode is exhausting, especially for your immune system, which can become suppressed due to chronic stress. It not only can keep your guard down against new disease, but also exacerbate existing conditions.
Now that you know what stress is doing to your body, use the most researched stress reduction technique: meditation. It’s not all Buddha statues and incense. In fact, meditation is simply an umbrella term for practices like mindfulness, mantras, guided meditation, yoga, and deep breathing techniques, which research shows can do everything from reduce stress to promote weight loss and stave off diseases such as Alzheimer’s, cancer, and high blood pressure.
We know, we know. But you’re stressed now! Try Dr. Weil’s trusted 4:7:8 breathing exercise, what he calls a “natural tranquilizer” for your nervous system.
This article was republished with permission from Health Holistic Living.
The post How extreme stress affects your health appeared first on Just Naturally Healthy.
Statin cholesterol-lowering drugs are widely touted as the best way to lower your cholesterol and thereby prevent a heart attack. They’re recommended to people who have “high cholesterol,” those who have heart disease, and even for some healthy people as a form of preventive medicine.
Statins are among the most widely prescribed drugs on the market, with more than 1 in 4 Americans over 45 taking them. This already inflated number is set to increase significantly due to draft recommendations issued earlier this year by the U.S. Preventive Services Task Force (USPSTF).
This federal advisory board recommended statin treatment for people between the ages of 40 and 75 with a 10 percent or greater risk of heart problems in the next 10 years (based on the 2013 AHA-ACC online calculator1) — even if they have not had a previous heart attack or stroke.
Needless to say, if you’re a U.S. adult aged 40 or beyond, there’s a good chance your doctor may bring up statins at your next visit, so you need to do your homework to determine if these drugs are truly right for you — and there’s a good chance they’re not.
Statin drugs work to lower cholesterol, and as your levels fall, you may assume that is proof that you’re getting healthier and lowering your risk of heart disease and heart attack. But that would be far from the truth.
There is far more that goes into your risk of heart disease than your cholesterol levels. Further, there is evidence showing that statins may actually make your heart health worse and only appear effective due to statistical deception.
One report published in the Expert Review of Clinical Pharmacology concluded that statin advocates used a statistical tool called relative risk reduction (RRR) to amplify statins’ trivial beneficial effects.2
If you look at absolute risk, statin drugs benefit just 1 percent of the population. This means that out of 100 people treated with the drugs, one person will have one less heart attack. This doesn’t sound so impressive, so statin supporters use a different statistic called relative risk.
Just by making this statistical sleight of hand, statins suddenly become beneficial for 30 to 50 percent of the population. As STATS at George Mason University explained, “An important feature of relative risk is that it tells you nothing about the actual risk.”3
Statins deplete your body of coenzyme Q10 (CoQ10), which accounts for many of their devastating results. Although it was proposed to add a black box warning to statins stating this, the U.S. Food and Drug Administration (FDA) decided against it in 2014.
CoQ10 is used for energy production by every cell in your body, and is therefore vital for good health, high energy levels, longevity, and general quality of life. CoQ10’s reduced form, ubiquinol, is a critical component of cellular respiration and production of adenosine triphosphate (ATP).
ATP is a coenzyme used as an energy carrier in every cell of your body. When you consider that your heart is the most energy-demanding organ in your body, you can surmise how potentially devastating it can be to deplete your body’s main source of cellular energy.
So while one of statins’ claims to fame is warding off heart disease, you’re actually increasing your risk when you deplete your body of CoQ10. The depletion of CoQ10 caused by the drug is why statins can increase your risk of acute heart failure.
So if you’re taking a statin drug, you MUST take Coenzyme Q10 as a supplement. If you’re over 40, I would strongly recommend taking ubiquinol instead of CoQ10, as it’s far more effectively absorbed by your body.
In every study conducted so far, ubiquinol has been shown to be far more bioavailable than the non-reduced form (CoQ10). Dr. Steven Sinatra,cardiologist and founder of the New England Heart Center, recommends taking at least 100 milligrams (mg), but preferably 200 mg of high-quality CoQ10 or ubiquinol daily.
One study in the European Journal of Pharmacology showed that ubiquinol effectively rescued cells from the damage caused by the statin drug simvastatin, thereby protecting muscle cells from myopathies.4
The other part most people don’t realize is that CoQ10 and ubiquinol are lipid-soluble materials biosynthesized in your blood. The carrier is the blood lipid cholesterol.
The ubiquinol actually keeps your LDL (often referred to as the “bad” cholesterol) reduced, as it’s an exceptionally potent antioxidant.
Reduced LDL cholesterol isn’t bad cholesterol at all. Only the oxidized version will cause a problem. So by reducing CoQ10 production in your body, you’re also removing the mechanism that keeps your LDL cholesterol from doing harm in your body.
A new finding was published in March 2015, and it is not yet widely known.
Research published in Expert Review of Clinical Pharmacology revealed that, in contrast to the current belief that cholesterol reduction with statins decreases atherosclerosis, the drugs may instead actually stimulate atherosclerosis and heart failure.5
There were several physiological mechanisms discussed in the study that show how statin drugs may make your heart health worse, one being that they inhibit the synthesis of vitamin K2. Vitamin K2 protects your arteries from calcification. Without it, plaque levels worsen.
Vitamin K2’s biological role is to help move calcium into the proper areas in your body, such as your bones and teeth. It also plays a role in removing calcium from areas where it shouldn’t be, such as in your arteries and soft tissues.
According to a 2009 Dutch study, vitamin K2 is associated with reduced vascular calcification even at small dietary intakes.6
Statin drugs inhibit the function of vitamin K2 in your body, which means taking them may put you at risk of vitamin K2 deficiency, a condition known to contribute to a number of chronic diseases, including:
| Osteoporosis | Heart disease | Heart attack and stroke |
| Inappropriate calcification, from heel spurs to kidney stones | Brain disease | Cancer |
Statins lower cholesterol by inhibiting the enzyme in your liver that produces cholesterol (HMG coenzyme A reductase). Unfortunately this is the same enzyme that produces not only CoQ10 but also ketones, which are crucial nutrients to feed your mitochondria.
Ketones are vitally important biological signaling molecules. There are three ketone bodies, acetoacetate, beta hydroxybutyrate, and acetone.
They’re produced in your liver (they’re byproducts of the breakdown of fatty acids) and production increases during fasting.7 As noted in the journal Trends in Endocrinology & Metabolism:8
“Ketone bodies are emerging as crucial regulators of metabolic health and longevity, via their ability to regulate HDAC [histone deacetylases] activity and thereby epigenetic gene regulation.”
Ketone bodies appear to inhibit HDAC function, which is implicated in the regulation of aging. Further, researchers noted “ketone bodies may link environmental cues such as diet to the regulation of aging.”9
Because statins deplete your body of CoQ10, inhibit synthesis of vitamin K2, and reduce the production of ketone bodies, they increase your risk of other serious diseases. This includes:
Cancer
Research has shown that long-term statin use (10 years or longer) more than doubles women’s risk of two major types of breast cancer: invasive ductal carcinoma and invasive lobular carcinoma.10 According to Dr. Sinatra, statins block the squalene pathway (squalene is the precursor to cholesterol), which he believes is essential in preventing breast cancer.
In addition, the use of any statin drug, in any amount, was associated with a significantly increased risk for prostate cancer in a separate study, and there was an increasing risk that came along with an increasing cumulative dose.11
According to a letter to the editor published in the Journal of Clinical Oncology:12
“Several cholesterol-lowering drugs, including statins, have been found to be carcinogenic in rodents in doses that produce blood concentrations of the drugs similar to those attained in treating patients.
In accordance, breast cancer occurred in 12 of 286 women in the treatment group of the CARE (Cholesterol and Recurrent Events) trial, but only in one of 290 in the placebo group … In the PROSPER (Prospective Study of Pravastatin in the Elderly at Risk) trial, cancer occurred in 245 of 2,891 patients in the treatment group, but only in 199 of 2,913 in the placebo group …
In the SEAS (Simvastatin and Ezetimibe in Aortic Stenosis) trial, cancer occurred in 39 of 944 patients in the treatment group, but only in 23 of 929 in the placebo group …
In the two first simvastatin trials, nonmelanoma skin cancer was seen more often as well, and with statistical significance if the results are calculated together … The latter finding may explain the current so-called epidemic of nonmelanoma skin cancer.”
Diabetes
Statins have also been shown to increase your risk of diabetes via a number of different mechanisms. The most important one is that they increase insulin resistance, which can be extremely harmful to your health. Secondly, statins increase your diabetes risk by raising your blood sugar. Statins work by preventing your liver from making cholesterol.
As a result, your liver returns the sugar to your bloodstream, which raises your blood sugar levels. These drugs also rob your body of certain valuable nutrients, which can also impact your blood sugar levels. Two nutrients in particular, vitamin D and CoQ10, are both needed to maintain ideal blood glucose levels. A 2011 meta-analysis confirmed the higher the dosage of statin drugs being taken, the greater the diabetes risk.
The “number needed to harm” for intensive-dose statin therapy was 498 for new-onset diabetes — that’s the number of people who need to take the drug in order for one person to develop diabetes.13 In even simpler terms, 1 out of every 498 people who are on a high-dose statin regimen will develop diabetes.
The following scientific reviews also reached the conclusion that statin use is associated with increased incidence of new-onset diabetes:
The study included data from more than 345,400 patients over a period of two years. On average, statins increased fasting plasma glucose in non-diabetic statin users by 7 mg/dL, and in diabetics, statins increased glucose levels by 39 mg/dL.
Neurodegenerative Diseases
Cholesterol is also essential for your brain, which contains about 25 percent of the cholesterol in your body. It is critical for synapse formation, i.e. the connections between your neurons, which allow you to think, learn new things, and form memories. So perhaps it’s not surprising that memory loss is widely reported in association with statin use.
Further, remember that statins reduce ketone production. Ketone bodies are used as fuel by your brain, and they have also demonstrated the capacity to protect against neuronal disease, seizures, and age-related brain diseases, such as Alzheimer’s, Huntington’s, and Parkinson’s. Researchers from Penn State College of Medicine even found statins were associated with an increased Parkinson’s risk.16
High total cholesterol and LDL were also associated with a lower risk of Parkinson’s disease. The study concluded, “Statin use may be associated with a higher PD [Parkinson’s disease] risk, whereas higher total cholesterol may be associated with lower risk.”
Musculoskeletal Disorders
Statin users are more likely to suffer from musculoskeletal conditions, injuries and pain than non-users.17 Myalgia, muscle weakness, muscle cramps, rhabdomyolysis, autoimmune muscle disease, and tendinous diseases have all been reported in association with statin use.
One reason for this may be statins’ interference with selenium-containing proteins. Selenoproteins such as glutathione peroxidase are crucial for preventing oxidative damage in your muscle tissue. As reported by Wellness Resources:18
“Blocking the selenoprotein enzyme glutathione peroxidase is akin to pouring gasoline on the fire of inflammation and free radicals, which damages muscle tissue. In fact, the scientists described this blocking of the selenoproteins reminiscent of selenium deficiency induced heart failure, known as Keshan’s disease first identified in the 1930s.”
Further, according to a study published in JAMA Internal Medicine:19
“ … [S]tatin use is associated with an increased likelihood of diagnoses of musculoskeletal conditions, arthropathies, and injuries … Several factors may explain the musculoskeletal AEs [adverse events] of statin therapy, including the inhibitory effect on coenzyme Q10 synthesis, selenoprotein synthesis, and the mitochondrial respiratory chain.
In addition, in vitro studies indicated that statins may affect apoptosis genes; misregulation of apoptosis is associated with myopathy. Pathologic studies also have shown that statin use may be associated with myopathy in the presence of normal creatine kinase levels, even in the absence of symptoms.
Statin-associated necrotizing autoimmune myopathy was noted to persist or progress despite cessation of statin therapy.”
Cataracts
An objective review of PubMed, EMBASE, and Cochrane review databases found that for every 10,000 people taking a statin, there were 307 extra patients with cataracts.20 This was supported by a separate JAMA study, which further revealed that the risk of cataracts is increased among statin users compared with non-users.21 Cataract is a clouding of your eye lens and is a main cause of low vision among the elderly.
If you decide to take a statin, a vitamin K2 supplement is highly recommended. MK-7 is the form you’ll want to look for in supplements; it’s extracted from the Japanese fermented soy product called natto. Professor Cees Vermeer, one of the world’s top vitamin K2 researchers, recommends between 45 mcg and 185 mcg daily for adults.
You must use caution on the higher doses if you take anticoagulants, but if you are generally healthy and not on these types of medications, I suggest 150 mcg daily. You’ll also need to make sure you take CoQ10 or ubiquinol (the reduced form) with it. One study evaluated the benefits of CoQ10 and selenium supplementation for patients with statin-associated myopathy.22
Compared to those given a placebo, the treatment group experienced significantly less pain, decreased muscle weakness and cramps, and less fatigue.
Are you looking for a non-drug way to boost your heart health? Here are some of my top recommendations:
The post The Case for Not Taking A Statin appeared first on Just Naturally Healthy.
I have a few “candy corn” toenails, meaning toenail fungus, on the big toe and baby toe. I used to have it on both feet and successfully treated it on the other one with tea tree oil. For whatever reason, that [remedy] hasn’t been successful with the nails on this foot. I’ve even clipped the nails very far down in order to get the tea tree oil to the fungus-y hotspot. It’s almost to the base of my toenail on the big toe, and has already reached the base of the nail on my baby toe! Is there any hope of being able to wear peep-toe shoes again? I have such jealousy of perfectly manicured toes!
There are many ways to treat toenail fungus successfully. I feel that both internal and external treatment are necessary to be totally successful. Either way, both still take several months to be effective.
The dose depends on the individual’s weight. Follow a low-sugar, low-carbohydrate, anti-candida/antifungal diet for three weeks (known as the Caveman Diet).
Do this three times per week. You should notice a new, clean nail growing back to replace the damaged nail. Continue treatment for 6 to 8 weeks.
The post Get Rid of Toenail Fungus Naturally appeared first on Just Naturally Healthy.
Baku has renewed its contract to host Formula One's Azerbaijan Grand Prix through 2023
Italian car brand Alfa Romeo will be the only name on its Formula One team partnership with Switzerland-based Sauber this season
We all know that sitting for long periods of time is not good for our body, but is it deadly? Recent studies show that sitting for hours leads to a whole host of chronic illness, regardless of your age.
Think about how many hours in the day you sit. Driving to work, sitting at work, eating your meals, watching TV.
Because I work from home I miss the commute to work. I also have a dog who gets me out walking throughout the day. Often times I will do a load of laundry or dishes during my day, yet from all the studies I have read I still sit way to long during my day. Since I also suffer with fibromyalgia I know only too well that sitting in one position for too long will cause me pain and suffering so I do tend to move around a lot during the day, but some days I will get into a project or writing recipes on the computer and before I know it I have been sitting way longer than I had planned.
50-70 percent of people spend six or more hours a day sitting
Experts agree that being more active means moving during the day. So how are some of the ways you can ward off “sitting disease”? Here are some suggestions
Does your work place offer alternatives to sitting on the job? Many workplaces are now offering standing desks. The problem with standing desk or ergonomic chairs is that we are still staying in one position. Whether you are standing or sitting we need to move.
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